Programme(s) to which this project applies:
|☑ MPhil/PhD||☒ MRes[Med]||☒ URIS|
Systemic lupus erythematosus (SLE) is a prototypic autoimmune disorder that predominantly affect young females. Immunologically, SLE is characterized by a loss of tolerance to self-antigens, T and B cell activation, and elevated pathogenic autoantibody production. The aetiology of SLE is highly complex with interplay of genetic, hormonal and environmental factors. This also entails heterogeneity in clinical manifestations. Our team has previously identified phenotypic, functional abnormalities as well as the underlying mechanisms in various immune cellular compartments in SLE patients. Through understanding the underlying causes of cellular and molecular immunodysregulation in SLE, we aim to identify potential therapeutic targets for exploitation in clinical settings. Our current research projects focus on the following areas:
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